By Susan B. Waltzman, J. Thomas Roland
The moment variation of Cochlear Implants offers a finished assessment of the cutting-edge concepts for comparing and choosing the cochlear implant candidate. transparent descriptions of surgical strategies advisor the reader via implantation systems, and chapters handle vital matters reminiscent of speech construction, language improvement, and schooling in implant recipients.
This moment variation features:
- New chapters at the genetics of listening to loss, sound processing, binaural listening to, and electroacoustic stimulation
- Complete dialogue of the latest advances in review techniques, surgical procedure, programming equipment, speech processing options, and extra
- Precise, easy-to-follow tables and figures increase comprehension of the elemental technology, learn and scientific techniques coated in the textual content
- Coverage of the scientific and surgical issues of cochlear implantation
- Insights from an interdisciplinary staff of specialists in otolaryngology, audiology, the fundamental sciences, speech pathology, and education
Ideal for studying and reference, Cochlear Implants synthesizes the foremost details wanted by way of practitioners, researchers, and scholars in a number disciplines. Readers will take advantage of either the scope and thoroughness of this authoritative reference.
Dr. Roland commemorated in top medical professionals 2012 factor of ny Magazine
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Additional resources for Cochlear Implants
Although it does not feature in the acronym, hearing loss is a common finding in MELAS. Large kindreds and patient series have been reported, demonstrating that over 50% of patients have moderate or severe SNHL: 21 of 28 Australian patients with MELAS in a series were deaf, as were eight of 14 British patients in a series (Morgan-Hughes et al, 1995; Sue et al, 1998). Three constraints control the phenotypic expression of the mutation: the percentage of mutant mitochondrial DNA in the target tissue (though this percentage carries at most a loose correlation with clinical lesions), the degree of exposure to oxidative stress of different organs or cell populations, and unidentified collaborating somatic mutations that selectively enhance aspects of the syndrome.
1996). Genes responsible for human hereditary deafness: symphony of a thousand. Nat Genet 14:385 – 391 Poissant SF, Megerian CA, Hume D. (2003). Cochlear implantation in a patient with neurofibromatosis type 1 and profound hearing loss: evidence to support a cochlear site of lesion. Otol Neurotol 24:751–756 Prezant RT, Shohat M, Jaber L, Pressman S, Fischel-Ghodsian N. (1992). Biochemical characterization of a pedigree with mitochondrially inherited deafness. Am J Med Genet 44:465 – 472 Rance G, Beer DE, Cone-Wesson B, et al.
Were found in six of 12 patients with mild to moderate deafness, and after excluding severe and profoundly deaf patients with a complete absence of responses, there were normal and symmetric brainstem evoked responses in 18 of 20 latencies recorded from 10 patients. Promontory stimulation testing in two patients was normal, and CT and MRI were reported as showing no lesions that could contribute to hearing loss. In addition, central auditory lesions have been reported as a cause of hearing loss in MELAS.