Download An Introduction to Radiation Protection 6E by Alan Martin, Sam Harbison, Karen Beach, Peter Cole PDF

By Alan Martin, Sam Harbison, Karen Beach, Peter Cole

A brand new variation of the UK's usual creation to the risks provided through ionizing radiation and the tools of safeguard and control.

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Extra info for An Introduction to Radiation Protection 6E

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2. Stochastic effects, in which the probability of occurrence of the effect increases with dose. 7). 1 Acute radiation effects The harmful tissue reactions that arise from acute radiation exposure (a large dose over a relatively short period of time) are those that occur within a few weeks after the receipt of the dose. The effects result from a major depletion of cell populations in a number of body organs caused by the killing of cells and the prevention or delay of cell division. The main effects are attributable to bone marrow, gastrointestinal or neuromuscular damage depending on the dose received.

B radiation causes ionization of a similar density to g radiation and so its weighting factor is also 1. 5–20* a particles, fission fragments 20 *Depending on energy. Submultiples for thermal neutrons to 20 for fast neutrons. For a and other multiply charged particles, wR is also taken as 20. 1. 0002 Gy. What is his total equivalent dose? 019 Sv In the remainder of the book, we generally refer to equivalent dose simply as dose, except where this could lead to confusion. 5 EFFECTIVE DOSE A further complication is that different organs and tissues have differing sensitivities to radiation.

Clearly, only that exposure which occurs up to the time of conception can affect the genetic characteristics of the offspring and, since the mean age of childbearing is about 30 years, only a proportion of the dose received by a typical population will be genetically harmful. 2 ¥ 10−2/Sv. 1 ¥ 10−2/Sv. 8 DETRIMENT To assist in quantifying and combining the consequences of exposure of different organs and tissues of the body, the ICRP has developed the concept of detriment. This takes into account the relative risks and the average latency period of fatal cancers in different organs, an allowance for the ill health resulting from non-fatal cancers and for the risk of serious heritable effects in all future generations descended from an exposed individual.

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